Development of skin lesions in filaggrin-deficient mice is dependent on adaptive immunity - 30/03/13
Doi : 10.1016/j.jaci.2012.12.1576
Sabine Leisten, MSc a, b, Michiko K. Oyoshi, PhD a, Claire Galand, PhD a, Jason L. Hornick, MD, PhD c, Michael F. Gurish, PhD d, Raif S. Geha, MD a 
a Division of Immunology, Boston Children’s Hospital, Boston, Mass
b Department of Biology, University of Hamburg, Germany
c Department of Pathology, Brigham and Women’s Hospital, Boston, Mass
d Division of Rheumatology, Immunology and Allergy, Brigham and Women’s Hospital, Boston, Mass
| This work was supported by grants from the Atopic Dermatitis Research Network (NIH/NIAID contract number HHSN272201000020C) and NIH (R01 AR047417). S.L. was supported by a PhD scholarship from the German Academic Exchange Service (DAAD). M.K.O. was supported by the Children’s Hospital Boston Faculty Career Development Fellowship/Eleanor and Miles Shore Program for Scholars in Medicine at Harvard Medical School. |
|
| Disclosure of potential conflict of interest: R. S. Geha has received one or more grants from or has one or more grants pending with the National Institutes of Health (NIH). M. F. Gurish has been supported by one or more grants from the NIH and has received royalties from UpToDate, Inc. S. Leisten has been supported by the German Academic Exchange Service. M. K. Oyoshi has been supported by the Boston Children’s Hospital Faculty Career Development Fellowship/Eleanor and Miles Shore Program for Scholars in Medicine at Harvard Medical School. The rest of the authors declare that they have no relevant conflicts of interest. |
© 2013
American Academy of Allergy, Asthma & Immunology. Publié par Elsevier Masson SAS. Tous droits réservés.
Vol 131 - N° 4
P. 1247 - avril 2013 Retour au numéro
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