Effect of losartan versus atenolol on aortic valve sclerosis (a LIFE substudy) - 26/08/11
Doi : 10.1016/j.amjcard.2004.06.074
Michael H. Olsen, MD, PhD a, ⁎ 
, Kristian Wachtell, MD, PhD a, Jonathan N. Bella, MD b, Jennifer E. Liu, MD b, Kurt Boman, MD c, Eva Gerdts, MD, PhD d, Vasilios Papademetriou, MD e, Markku S. Nieminen, MD f, Jens Rokkedal, MD a, Björn Dahlöf, MD g, Richard B. Devereux, MD b
, Kristian Wachtell, MD, PhD a, Jonathan N. Bella, MD b, Jennifer E. Liu, MD b, Kurt Boman, MD c, Eva Gerdts, MD, PhD d, Vasilios Papademetriou, MD e, Markku S. Nieminen, MD f, Jens Rokkedal, MD a, Björn Dahlöf, MD g, Richard B. Devereux, MD b a Glostrup University Hospital, Glostrup, Denmark
b Weill Medical College of Cornell University, New York, New York, USA
c Skellefteå Lasarett, Skellefteå, Sweden
d Haukeland Hospital, Bergen, Norway
e Veterans Affairs Medical Center, Washington, DC, USA
f Helsinki University Hospital, Helsinki, Finland
g Sahlgrenska University Hospital-Östra, Göteborg, Sweden
*Dr. Olsen's address is: Department of Clinical Physiology and Nuclear Medicine, Glostrup University Hospital, DK-2600 Glostrup, Denmark
Résumé |
Neither losartan- nor atenolol-based antihypertensive regimens could prevent the progression of aortic valve (AV) sclerosis in elderly, high-risk hypertensive patients, and the regression of AV sclerosis did not translate into reduced cardiovascular risk.
Le texte complet de cet article est disponible en PDF.| This study was supported in part by grants from Merck & Co., Inc., West Point, Pennsylvania. |
© 2004
Excerpta Medica Inc. Tous droits réservés.
Vol 94 - N° 8
P. 1076-1080 - octobre 2004 Retour au numéro
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