Pediatric hypereosinophilic syndrome (HES) differs from adult HES - 15/08/11
Doi : 10.1016/j.jpeds.2004.09.014
Hary T. Katz, MD, S. Jaharul Haque, PhD, Fred H. Hsieh, MD ⁎ 
From the Department of Pulmonary, Allergy, and Critical Care Medicine and the Department of Cancer Biology, Cleveland Clinic Foundation, Cleveland, Ohio
Reprint requests: Dr Fred H. Hsieh, Cleveland Clinic Foundation, Pulmonary, Allergy, and Critical Care Medicine, Allergy and Immunology Section, 9500 Euclid Avenue C22, Cleveland, OH 44195.Résumé |
The idiopathic hypereosinophilic syndrome (HES) developed in a 15-year-old boy who presented with colitis, cough, rash, and hepatitis. Molecular analysis failed to demonstrate the Fip1-like1-Platelet Derived Growth Factor Receptor ⍺ chain (FIP1L1-PDGFRA) mutation described in adult patients with HES. There are significant clinical differences between the pediatric and adult presentations of HES.
Le texte complet de cet article est disponible en PDF.Mots-clés : FIP1L1-PDGFRA, HES
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| Supported by NIH grants HL074129-01, CA095006-02, and GM060533-05. |
© 2005
Elsevier Inc. Tous droits réservés.
Vol 146 - N° 1
P. 134-136 - janvier 2005 Retour au numéro
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