An integrated model of alopecia areata biomarkers highlights both TH1 and TH2 upregulation - 06/11/18
Doi : 10.1016/j.jaci.2018.06.029
Teresa Song, BS a, b, Ana B. Pavel, PhD a, Huei-Chi Wen, MD, PhD a, Kunal Malik, MD a, b, Yeriel Estrada, BS a, Juana Gonzalez, PhD c, Peter W. Hashim, MD, MHS a, John K. Nia, MD a, Danielle Baum, RN a, Grace Kimmel, MD a, Giselle K. Singer, BS a, James G. Krueger, MD, PhD c, Emma Guttman-Yassky, MD, PhD a 
a Department of Dermatology, Laboratory of Inflammatory Skin Diseases, Icahn School of Medicine at Mount Sinai, New York, NY
b College of Medicine, SUNY Downstate, New York, NY
c Laboratory for Investigative Dermatology, Rockefeller University, New York, NY
| Disclosure of potential conflict of interest: J. G. Krueger has received research support (grants paid to his institution) and/or personal fees from Pfizer, Amgen, Janssen, Lilly, Merck, Novartis, Kadmon, Dermira, Boehringer, Innovaderm, Kyowa, BMS, Serono, BiogenIdec, Delenex, AbbVie, Sanofi, Baxter, Paraxel, Xenoport, and Kineta. E. Guttman-Yassky is an employee of Mount Sinai and has received research funds (grants paid to the institution) from: Abbvie, Celgene, Eli Lilly, Janssen, Medimmune/Astra Zeneca, Novartis, Pfizer, Regeneron, Vitae, Glenmark, Galderma, Asana, Innovaderm, Dermira, and UCB and is also a consultant for Sanofi Aventis, Regeneron, Stiefel/GlaxoSmithKline, MedImmune, Celgene, Anacor, AnaptysBio, Dermira, Galderma, Glenmark, Novartis, Pfizer, Vitae, Leo Pharma, Abbvie, Eli Lilly, Kyowa, Mitsubishi Tanabe, Asana Biosciences, and Promius. The rest of the authors declare that they have no relevant conflicts of interest. |
© 2018
American Academy of Allergy, Asthma & Immunology. Publié par Elsevier Masson SAS. Tous droits réservés.
Vol 142 - N° 5
P. 1631 - novembre 2018 Retour au numéro
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