Aspergillosis, eosinophilic esophagitis, and allergic rhinitis in signal transducer and activator of transcription 3 haploinsufficiency - 06/09/18
Doi : 10.1016/j.jaci.2018.05.009
Mukil Natarajan, MD a, Amy P. Hsu, BA a, ∗, Michael A. Weinreich, MD, PhD b, ∗, Yuan Zhang, PhD b, Julie E. Niemela, MS c, John A. Butman, MD, PhD d, Stefania Pittaluga, MD e, Janyce Sugui, PhD a, Amanda L. Collar, BS a, Jean K. Lim, PhD f, Tirdad Zangeneh, DO g, Tara Carr, MD g, Andrew J. Oler, PhD h, Morgan Similuk, ScM a, Lindsey B. Rosen, BS a, Jigar V. Desai, PhD a, Alexandra F. Freeman, MD a, Steven M. Holland, MD a, Kyung J. Kwon-Chung, PhD a, Joshua D. Milner, MD b, Michail S. Lionakis, MD, ScD a 
a Laboratory of Clinical Immunology and Microbiology, National Institute of Allergy and Infectious Diseases (NIAID), National Institutes of Health (NIH), Bethesda, Md
b Laboratory of Allergic Diseases, NIAID, National Institutes of Health (NIH), Bethesda, Md
c Department of Laboratory Medicine, Clinical Center (CC), National Institutes of Health (NIH), Bethesda, Md
d Radiology and Imaging Sciences, CC, National Institutes of Health (NIH), Bethesda, Md
e Laboratory of Pathology, Center for Cancer Research, National Cancer Institute, National Institutes of Health (NIH), Bethesda, Md
f Department of Microbiology, Icahn School of Medicine at Mount Sinai, New York, NY
g Department of Medicine, University of Arizona College of Medicine, Tucson, Ariz
h Bioinformatics and Computational Biosciences Branch, NIAID, NIH, Bethesda, Md
| This work was supported by the Intramural Research Program of the Division of Intramural Research, National Institute of Allergy and Infectious Diseases, National Institutes of Health and by the Deputy Director of Intramural Research through the Clinical Center Genomics Opportunity. |
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| Disclosure of potential conflict of interest: The authors have no relevant conflicts of interest. |
© 2018
Publié par Elsevier Masson SAS.
Vol 142 - N° 3
P. 993 - septembre 2018 Retour au numéro
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