Clinical Neuropharmacology of Behavioral Recovery Following Brain Injury - 06/07/18
Doi : 10.1016/S1047-9651(18)30295-X
Robert L. Harmon, MD, MS a, ⁎, Michael G. Boyeson, PhD b
a From the Department of Physical Medicine and Rehabilitation, Medical College of Ohioat Toledo, Toledo, Ohio
b Private practice, Waunakee, Wisconsin
*Address reprint requests to: Robert L. Harmon, MD, MS, Department of Physical Medicine and Rehabilitation, Medical College of Ohio at Toledo, P.O. Box 10008, Toledo, OH 43699.Department of Physical Medicine and RehabilitationMedical College of Ohio at ToledoP.O. Box 10008ToledoOH43699
Résumé |
Of the many models that have been offered to explain recovery of particular behaviors after various types of brain injury, all are based on the observation that a significant amount of behavioral recovery occurs in the absence of complete regrowth of brain tissue. Altering biochemical events, such as controlling the massive release of neurotransmitters or reducing edema and lactic acid after brain injury, may provide more specific methods to intercede in the injury process before the damage has progressed to its final, more permanent state.
Le texte complet de cet article est disponible en PDF.© 1997
Elsevier Inc. Tous droits réservés.
Vol 8 - N° 4
P. 651-665 - novembre 1997 Retour au numéro
Article précédent
- Drugs in the Management of Cute Traumatic Brain Injury
- Ramesh Raghupathi, Tracy K. McIntosh
- Drugs for Medical Concerns in Traumaticbrain Injury
- Flora M. Hammond, Kertia L. Black
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