GITR cosignal in ILC2s controls allergic lung inflammation - 04/05/18
Doi : 10.1016/j.jaci.2018.01.028
Hiroyuki Nagashima, PhD a, ∗, Yuko Okuyama, PhD a, ∗, Tsuyoshi Fujita, MD, PhD a, ∗, Takeo Takeda, MSc a, Yasutaka Motomura, PhD b, Kazuyo Moro, PhD b, Takanori Hidaka, MD, PhD c, Koki Omori, MSc a, Tsuyoshi Sakurai, MD a, Tomoaki Machiyama, MD a, Lishomwa C. Ndhlovu, MD, PhD d, Carlo Riccardi, PhD e, Takanori So, PhD a, f, Naoto Ishii, MD, PhD a 
a Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan
b Laboratory for Innate Immune Systems, RIKEN Center for Integrative Medical Sciences, Yokohama, Japan
c Department of Dermatology, Tohoku University Graduate School of Medicine, Sendai, Japan
d Department of Tropical Medicine, Hawaii Center for AIDS, John A. Burns School of Medicine, University of Hawaii, Honolulu, Hawaii
e Section of Pharmacology, Department of Medicine, University of Perugia, Perugia, Italy
f Laboratory of Molecular Cell Biology, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Toyama, Japan
| This study was supported in part by JSPS KAKENHI (grant nos. 16K15508 and 17K08875), the Yamaguchi Educational and Scholarship Foundation (T.S.), and the Daiichi-Sankyo Foundation of Life Science (T.S.). |
|
| Disclosure of potential conflict of interest: Y. Okuyama and N. Ishii received a grant from JSPS for this work. T. So received grants from Yamaguchi Educational and Scholarship Foundation and Daiichi-Sankyo Foundation of Life Science for this work. The rest of the authors declare that they have no relevant conflicts of interest. |
© 2018
Publié par Elsevier Masson SAS.
Vol 141 - N° 5
P. 1939 - mai 2018 Retour au numéro
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