ORMDL deregulation increases stress responses and modulates repair pathways in Drosophila airways - 13/05/15
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This work was supported by the German Research Foundation, DFG, SFB/Transregio 22, project A07. |
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Disclosure of potential conflict of interest: K. Kallsen has received research support from the German Research Foundation (SFB/Transregio 22 [SFB/TR22]—funding of PhD position and material costs), has received travel scholarships from the German Society for Immunology (DGfI) and Verein zur Erforschung infektiologischer und allergischer Prozesse e.V. (VEIAP), and is employed by Boehringer Ingelheim Pharma GmbH & Co. KG. B. Lindner has received research support and travel support from the German Research Foundation (SFB/TR22). M. Kabesch has received research support from the European Union, the German Ministry of Education and Research, and the German Research Foundation and has received lecture fees from the European Respiratory Society, the European Academy of Allergy and Clinical Immunology, the American Thoracic Society, Novartis, and Glaxo. H. Heine has received research support and travel support from the German Research Foundation (SFB/TR22). T. Roeder has received research support from the German Research Foundation (SFB/TR22, TPA7). The rest of the authors declare that they have no relevant conflicts of interest. |
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